Mophological syndrome

Articles

• interface hepatitis
  25 May 2004

  piecemeal necrosis

• lymphoepithelial sialadenitis
  5 November 2007

  LESA, myoepithelial sialadenitis, MESA, salivary benign lymphoepithelial lesion,
  Definition: LESA is found in virtually all patients with Sjögren’s syndrome but may occur without Sjogren’s syndrome. It is associated with lymphoid infiltration of salivary gland tissue, producing atrophy and destruction of the acini.
  Myoepithelial sialadenitis of the salivary gland is associated with an increased risk of lymphoma, but criteria that predict its development have not been clearly defined. (...)

• acute inflammation
  1 June 2004

  Digital cases
  Case 25 (HPC:25) : Acute appendicitis
  Case 94 (HPC:94) : Acute appendicitis
  Case 13 (HPC:13) : Acute synovitis
  Definition: Acute inflammation is a rapid response to an injurious agent that serves to deliver mediators of host defense-leukocytes and plasma proteins-to the site of injury.
  Types - Lesional patterns
  acute inflammation
  congestive inflammation
  cellular acute inflammation
  suppurative inflammation / suppuration
  abcess / abcesses / abcedation (...)

• colitis
  3 October 2003

  Images
  Pseudomembranous Colitis, Colectomy Specimen by Ed Euthman
  ipilimumab-induced colitis
  https://twitter.com/GIPathologyURMC/status/715943975526522880
  Lesional patterns
  acute colitis
  congestive colitis
  common acute colitis
  pseudomembranous colitis
  hemorrhagic colitis
  active colitis
  focal active colitis
  diffuse active colitis
  chronic colitis
  lymphocytic colitis (microscopical colitis )
  granulomatous colitis
  eosinophilic colitis
  GVHD-like colitis (...)

• giant cell transformation
  19 June 2003

  neonatal syncytial giant cell hepatitis, giant cell hepatitis, giant cell transformation of hepatocytes, hepatocytic giant cell transformation

• acute hepatitis
  30 May 2003

  Microscopy
  The essential components of the acute phase of hepatitis are:
  inflammatory-cell infiltration
  hepatocellular damage
  cholestasis
  Kupffer-cell activation
  endotheliitis
  bile-duct damage
  ductular reaction
  hepatocellular regeneration
  hepatocellular damage
  Hepatocellular damage range from minor degrees of cell swelling to cell death. They are accompanied by the inflammatory infiltration described below, reflecting the important role of cellular immunity in the (...)

• sclerosing cholangitis
  4 June 2003

  Sclerosing cholangitis can be primary (PSC) or secondary.
  Subtypes
  primary sclerosing cholangitis (PSC) neonatal sclerosing cholangitis auto-immune sclerosing cholangitis
  secondary sclerosing cholangitis (SSC) infectious chronic cholangitis biliary cryptosporidiosis (immunodeficiencies) Langerhans cell histiocytosis choledocholithiasis sickle cell disease total parenteral nutrition Hodgkin disease primary intestinal T-cell lymphoma (#9893108#) cystic fibrosis postbiliary surgery (...)

• ductopenia
  4 June 2003

  vanishing bile duct diseases, paucity of interlobular biliary ducts, ductopenic pattern, loss of interlobular bile ducts, bile duct loss, vanishing bile duct syndromes, hepatic ductopenia

• focally enhanced gastritis
  29 November 2004

  focal active gastritis

• canalicular cholestasis
  8 December 2004

  Canalicular cholestasis takes the form of bile plugs (bile thrombi) in dilated canaliculi.
  There is often brown or yellow pigment in nearby hepatocytes and Kupffer cells, but the distinction of this pigment from others such as lipofuscin and ceroid is not a serious practical problem; this is because the presence of bile in the canaliculi makes the diagnosis of cholestasis obvious.
  In general, cholestasis should only be diagnosed with great caution in the absence of bile plugs in (...)

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